2026-05-05T20:50:24Zhttps://uvadoc.uva.es/oai/request

oai:uvadoc.uva.es:10324/420572025-02-24T08:37:17Zcom_10324_1181com_10324_931com_10324_894col_10324_1387

León, Alberto José Gómez, Emma Garrote Adrados, José Antonio Bernardo Ordiz, David Barrera, Asterio Marcos, José Luis Fernández Salazar, Luis Ignacio Velayos Jiménez, Benito Blanco Quiros, Alfredo Arranz Sanz, Eduardo 2020-09-01T09:15:46Z 2020-09-01T09:15:46Z 2009 Mediators of Inflammation, Volume 2009, ID 580450, 10 pages 0962-9351 http://uvadoc.uva.es/handle/10324/42057 10.1155/2009/580450 1 10 Mediators of Inflammation 2009 1466-1861 Producción Científica Ulcerative colitis (UC) and Crohn’s disease (CD) are severeintestinal inflammatory conditions which result from adysregulated proinflammatory response to components ofthe gut microflora, inadequately controlled by normalregulatory/homeostatic mechanisms [1]. The expression ofproinflammatory cytokines is increased in inflammed biopsyareas from both UC and CD, and the effect of these cytokineson the intestine has been directly related with the inductionof tissue injury (destruction) [2,3] Ministerio de Sanidad, Consumo y Bienestar Social (PI020895, 02/3068) Junta de Castilla y León (VA057/04 and SAN1052-VA02/05) Ministerio de Educación y Ciencia (AP2002-1696) application/pdf eng Hindawi Publishing Corporation info:eu-repo/semantics/openAccess http://creativecommons.org/licenses/by-nc-nd/4.0/ © Hindawi Publishing Corporation Attribution-NonCommercial-NoDerivatives 4.0 Internacional Intestinos - Enfermedades Enfermedad de Chrom Enfermedad de Crohn Colitis ulcerosa 3205 Medicina Interna High levels of proinflammatory cytokines, but not markers of tissue injury, in unaffected intestinal areas from patients with IBD info:eu-repo/semantics/article info:eu-repo/semantics/publishedVersion https://www.hindawi.com/journals/mi/2009/580450/ SI