2024-03-29T04:39:34Zhttps://uvadoc.uva.es/oai/requestoai:uvadoc.uva.es:10324/58622021-06-23T11:41:24Zcom_10324_1179com_10324_931com_10324_894col_10324_1306
Pérez Castrillon, José Luis
San Miguel, Ángel
Vega, Gemma
Abad Manteca, Laura
Andrés Domingo, María
González Sagrado, Manuel
Luis Román, Daniel Antonio de
Dueñas Laita, Antonio
2009
Producción Científica
The atherosclerosis that appears in coronary, cerebrovascular and
peripheral arterial disease is responsible for most cardiovascular
diseases. It is characterized by chronic arterial inflammation caused
and exacerbated by disorders of the lipidic metabolism and other
clearly identified risk factors [1]. Calcification, which is initiated by an
active process in which inflammatory cytokines and other mediators
that regulate the phospho-calcium metabolism intervene, is characteristic
of atherosclerosis [2]. These mechanisms can intervene in an
opposite phenomenon that takes place at the level of the bone
characterized by a reduction in bone mineral content and alterations
in the microarchitecture that define osteoporosis. The association
between the two diseases, which share mechanisms but have a
different expression, is noteworthy.
application/pdf
http://uvadoc.uva.es/handle/10324/5862
eng
Elsevier Ireland Ltd.
Arteriosclerosis - Tratamiento
Cardiovascular, Aparato - Enfermedades
Levels of DKK1 in patients with acute myocardial infarction and response to atorvastatin
info:eu-repo/semantics/article
TEXT
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