2024-03-28T08:03:51Zhttps://uvadoc.uva.es/oai/requestoai:uvadoc.uva.es:10324/58362021-06-23T11:41:38Zcom_10324_1179com_10324_931com_10324_894col_10324_1306
Antibodies against Chlamydia pneumoniae and their relation to lymphocyte population levels
González Castañeda, Candelas
Pérez Castrillon, José Luis
Romero Gómez, Mercedes
Herreros Fernández, Vicente
Cardiovascular, Aparato, Efectos de los medicamentos sobre el
Atherosclerosis
Neumonia
Producción Científica
Chlamydia pneumoniae infection has long been suspected as a possible cause of atherosclerosis and has been frequently detected in
atheromatous plaques of the coronary arteries. Nevertheless, its distribution is not correlated to the severity or extent of the disease, but it
would support the hypothesis that the organism may be an active factor in the pathogenesis of atherosclerosis. A group of patients with
stable angina were examined as to whether or not the positivity of antibodies against Chlamydia pneumoniae modified cellular
populations as mechanisms responsible for the alterations of inflammatory response. We concluded that the presence of IgG anti-C.
pneumoniae antibodies do not participate in the activation of inflammatory mechanisms that may intervene in the genesis of
atherosclerosis in patients with stable angina.
2014-09-05T10:22:12Z
2014-09-05T10:22:12Z
2002
info:eu-repo/semantics/article
International Journal of Cardiology, 2002, vol. 82, p. 293-295
0167-5273
http://uvadoc.uva.es/handle/10324/5836
293
295
International Journal of Cardiology
82
eng
Attribution-NonCommercial-NoDerivatives 4.0 International
info:eu-repo/semantics/openAccess
http://creativecommons.org/licenses/by-nc-nd/4.0/
application/pdf
Elsevier Science Ireland Ltd.
https://uvadoc.uva.es/bitstream/10324/5836/6/CASTRILLON%2010%5b1%5d.pdf.jpg
Hispana
TEXT
http://creativecommons.org/licenses/by-nc-nd/4.0/
UVaDOC. Repositorio Documental de la Universidad de Valladolid
http://uvadoc.uva.es/handle/10324/5836