RT info:eu-repo/semantics/article
T1 Antibodies against Chlamydia pneumoniae in stable angina and interleukin-6 levels
A1 González Castañeda, Candelas
A1 Pérez Castrillon, José Luis
A1 Arranz Peña, Maria Luisa
A1 González, Victor
A1 Herreros Fernández, Vicente
K1 Neumonia
K1 Cardiovascular, Aparato - Enfermedades
AB Inflammation is a key mechanism in atherogenesis and the rapid progression of coronary artery disease. Tissue lesion occasions the release of chemical mediators, cytokines, accompanied by an increase in the blood concentrations of acute phase reactants, such as fibrinogen, C-reactive protein, serum amyloid A protein, sialic acid and ceruloplasmin and a reductionof those of albumin. It has been observed that these proteins are higher in patients with is-chemic heart disease and, furthermore, who have a higher tendency to present adverse cardiovascular incidents [1]. On the other hand, the inflammation appearsto be directly linked to the ‘vulnerability’ or ‘instability’ of the atheromatous plaques that pre-dispose to disruption and acute coronary incidents. The inflammatory mechanism, therefore, can repre-sent the final common connection channel of chronic infection between atherogenesis and the clinical manifestations of coronary artery disease [2].
PB Elsevier Science Ireland Ltd.
SN 0167-5273
YR 2003
FD 2003
LK http://uvadoc.uva.es/handle/10324/5837
UL http://uvadoc.uva.es/handle/10324/5837
LA eng
NO International Journal of Cardiology, 2003, vol. 88, p. 99-100
NO Producción Científica
DS UVaDOC
RD 27-nov-2024