The atherosclerosis that appears in coronary, cerebrovascular and
peripheral arterial disease is responsible for most cardiovascular
diseases. It is characterized by chronic arterial inflammation caused
and exacerbated by disorders of the lipidic metabolism and other
clearly identified risk factors [1]. Calcification, which is initiated by an
active process in which inflammatory cytokines and other mediators
that regulate the phospho-calcium metabolism intervene, is characteristic
of atherosclerosis [2]. These mechanisms can intervene in an
opposite phenomenon that takes place at the level of the bone
characterized by a reduction in bone mineral content and alterations
in the microarchitecture that define osteoporosis. The association
between the two diseases, which share mechanisms but have a
different expression, is noteworthy.
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