Sfoglia PRODUZIONE SCIENTIFICA per Autore "Tajada Esteban, Sendoa"

Items 1-17 di 17

A stochastic model of ion channel cluster formation in the plasma membrane

Sato, Daisuke; Hernández-Hernández, Gonzalo; Matsumoto, Collin; Tajada Esteban, Sendoa ; Moreno, Claudia M.; Dixon, Rose E.; O’Dwyer, Samantha; Navedo, Manuel F.; Trimmer, James S.; Clancy, Colleen E.; Binder, Marc D.; Santana, L. Fernando (2019)
BIN1 induces the formation of T-tubules and adult-like Ca2+ release units in developing cardiomyocytes

De La Mata Sampedro, Ana ; Tajada Esteban, Sendoa ; O'Dwyer, Samantha; Matsumoto, Collin; Dixon, Rose E.; Hariharan, Nirmala; Moreno, Claudia M.; Santana, Luis Fernando (2019)
Ca2+ entry into neurons is facilitated by cooperative gating of clustered CaV1.3 channels

Moreno, Claudia M; Dixon, Rose E; Tajada Esteban, Sendoa ; Yuan, Can; Opitz-Araya, Ximena; Binder, Marc D; Santana, Luis F (2016)
Calcium Permeable Channels in Cancer Hallmarks

Tajada Esteban, Sendoa ; Villalobos, Carlos (2020)
Cinnamaldehyde inhibits L-type calcium channels in mouse ventricular cardiomyocytes and vascular smooth muscle cells

Alvarez-Collazo, Julio; Alonso-Carbajo, Lucía; López-Medina, Ana I.; Alpizar, Yeranddy A.; Tajada Esteban, Sendoa ; Nilius, Bernd; Voets, Thomas; López López, José Ramón ; Talavera, Karel; Pérez García, María Teresa ; Alvarez, Julio L. (2014)
Distance constraints on activation of TRPV4 channels by AKAP150-bound PKCα in arterial myocytes

Tajada Esteban, Sendoa ; Moreno, Claudia M.; O’Dwyer, Samantha; Woods, Sean; Sato, Daisuke; Navedo, Manuel F.; Santana, L. Fernando (2017)
Down‐regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca2+ into hypertensive arterial smooth muscle

Tajada Esteban, Sendoa ; Cidad Velasco, María Del Pilar ; Colinas, Olaia; Santana, L. Fernando; López López, José Ramón ; Pérez García, María Teresa (2013)
Dynamic L-type CaV1.2 channel trafficking facilitates CaV1.2 clustering and cooperative gating

Ghosh, Debapriya; Nieves-Cintrón, Madeline; Tajada Esteban, Sendoa ; Brust-Mascher, Ingrid; Horne, Mary C.; Hell, Johannes W.; Dixon, Rose E.; Santana, Luis F.; Navedo, Manuel F. (2018)
Estudio del remodelado iónico en cáncer de colon mediante la transferencia mitocondrial y la inhibición de la síntesis de poliaminas.

Feijóo Salgado, Verónica (2024)
Función de los canales de K+ y Ca2+ en un modelo de hipertensión esencial

Tajada Esteban, Sendoa (2013)
Genetically engineered mice for combinatorial cardiovascular optobiology

Lee, Frank K; Lee, Jane C; Shui, Bo; Reining, Shaun; Jibilian, Megan; Small, David M; Jones, Jason S; Allan-Rahill, Nathaniel H; Lamont, Michael RE; Rizzo, Megan A; Tajada Esteban, Sendoa ; Navedo, Manuel F; Santana, Luis Fernando; Nishimura, Nozomi; Kotlikoff, Michael I (2021)
High blood pressure associates with the remodelling of inward rectifier K+ channels in mice mesenteric vascular smooth muscle cells

Tajada Esteban, Sendoa ; Cidad Velasco, María Del Pilar ; Moreno Domínguez, Alejandro; Pérez García, María Teresa ; López López, José Ramón (2012)
Kv1.3 channels can modulate cell proliferation during phenotypic switch by an ion-flux independent mechanism

Cidad Velasco, María Del Pilar ; Jiménez Pérez, Laura; García Arribas, Daniel; Miguel Velado, Eduardo; Tajada Esteban, Sendoa ; Ruiz McDavitt, Christian; López López, José Ramón ; Pérez García, María Teresa (2012)
Kv2.1 channels play opposing roles in regulating membrane potential, Ca 2+ channel function, and myogenic tone in arterial smooth muscle

O’Dwyer, Samantha C.; Palacio, Stephanie; Matsumoto, Collin; Guarina, Laura; Klug, Nicholas R.; Tajada Esteban, Sendoa ; Rosati, Barbara; McKinnon, David; Trimmer, James S.; Santana, L. Fernando (2020)
Total Internal Reflection Fluorescence Microscopy in Vascular Smooth Muscle

Nieves-Cintrón, Madeline; Tajada Esteban, Sendoa ; Santana, Luis Fernando; Navedo, Manuel F. (2018)
Transcriptional Basis of Ca2+ Remodeling Reversal Induced by Polyamine Synthesis Inhibition in Colorectal Cancer Cells

Pérez-Riesgo, Enrique; Hernando Pérez, María Elena ; Feijóo, Verónica; Tajada Esteban, Sendoa ; Núñez Llorente, Lucía ; Villalobos, Carlos (2023)
TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins

López López, José Ramón ; Alpizar, Yeranddy A.; Meseguer, Víctor; Enoch, Luis; Tajada Esteban, Sendoa ; Denlinger, Bristol; Fajardo, Otto; Manenschijn, Jan-Albert; Fernández Peña, Carlos; Talavera, Arturo; Kichko, Tatiana; Navia, Belén; Sánchez, Alicia; Señarís, Rosa; Reeh, Peter; Pérez García, María Teresa ; Voets, Thomas; Belmonte, Carlos; Talavera, Karel; Viana, Felix (2014)

Sfoglia PRODUZIONE SCIENTIFICA per Autore "Tajada Esteban, Sendoa"

A stochastic model of ion channel cluster formation in the plasma membrane ﻿

BIN1 induces the formation of T-tubules and adult-like Ca2+ release units in developing cardiomyocytes ﻿

Ca2+ entry into neurons is facilitated by cooperative gating of clustered CaV1.3 channels ﻿

Calcium Permeable Channels in Cancer Hallmarks ﻿

Cinnamaldehyde inhibits L-type calcium channels in mouse ventricular cardiomyocytes and vascular smooth muscle cells ﻿

Distance constraints on activation of TRPV4 channels by AKAP150-bound PKCα in arterial myocytes ﻿

Down‐regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca2+ into hypertensive arterial smooth muscle ﻿

Dynamic L-type CaV1.2 channel trafficking facilitates CaV1.2 clustering and cooperative gating ﻿

Estudio del remodelado iónico en cáncer de colon mediante la transferencia mitocondrial y la inhibición de la síntesis de poliaminas. ﻿

Función de los canales de K+ y Ca2+ en un modelo de hipertensión esencial ﻿

Genetically engineered mice for combinatorial cardiovascular optobiology ﻿

High blood pressure associates with the remodelling of inward rectifier K+ channels in mice mesenteric vascular smooth muscle cells ﻿

Kv1.3 channels can modulate cell proliferation during phenotypic switch by an ion-flux independent mechanism ﻿

Kv2.1 channels play opposing roles in regulating membrane potential, Ca 2+ channel function, and myogenic tone in arterial smooth muscle ﻿

Total Internal Reflection Fluorescence Microscopy in Vascular Smooth Muscle ﻿

Transcriptional Basis of Ca2+ Remodeling Reversal Induced by Polyamine Synthesis Inhibition in Colorectal Cancer Cells ﻿

TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins ﻿

A stochastic model of ion channel cluster formation in the plasma membrane

BIN1 induces the formation of T-tubules and adult-like Ca2+ release units in developing cardiomyocytes

Ca2+ entry into neurons is facilitated by cooperative gating of clustered CaV1.3 channels

Calcium Permeable Channels in Cancer Hallmarks

Cinnamaldehyde inhibits L-type calcium channels in mouse ventricular cardiomyocytes and vascular smooth muscle cells

Distance constraints on activation of TRPV4 channels by AKAP150-bound PKCα in arterial myocytes

Down‐regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca2+ into hypertensive arterial smooth muscle

Dynamic L-type CaV1.2 channel trafficking facilitates CaV1.2 clustering and cooperative gating

Estudio del remodelado iónico en cáncer de colon mediante la transferencia mitocondrial y la inhibición de la síntesis de poliaminas.

Función de los canales de K+ y Ca2+ en un modelo de hipertensión esencial

Genetically engineered mice for combinatorial cardiovascular optobiology

High blood pressure associates with the remodelling of inward rectifier K+ channels in mice mesenteric vascular smooth muscle cells

Kv1.3 channels can modulate cell proliferation during phenotypic switch by an ion-flux independent mechanism

Kv2.1 channels play opposing roles in regulating membrane potential, Ca 2+ channel function, and myogenic tone in arterial smooth muscle

Total Internal Reflection Fluorescence Microscopy in Vascular Smooth Muscle

Transcriptional Basis of Ca2+ Remodeling Reversal Induced by Polyamine Synthesis Inhibition in Colorectal Cancer Cells

TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins