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Por favor, use este identificador para citar o enlazar este ítem: http://uvadoc.uva.es/handle/10324/21820
Título: Non-steroidal anti-inflammatory drugs (NSAIDs) and neuroprotection in the elderly: a view from the mitochondria
Autor: Calvo Rodríguez, María
Villalobos, Carlos
Nuñez Llorente, Lucía
Año del Documento: 2015
Editorial: Medknow Publications
Descripción: Producción Científica
Documento Fuente: Neural Regen Res 10 (9), 1371-1372.
Resumen: The most important risk factor for stroke and neurodegeneration is aging. In fact, survival after stroke diminishes largely with aging. In fact, recovery after brain artery occlusion is dramatically worsened by aging, even normal aging is associated with neuron damage and cognitive decline. Mechanisms involved in aging-related, cognitive decline and susceptibility to neuron damage in stroke and neurodegeneration are largely unknown. One of the most important mechanisms contributing to neural dysfunction and death is excitotoxicity. This process is based on the fact that the excessive glutamate receptor stimulation may lead to neuronal damage. This overstimulation may be due to increased concentration of glutamate, or the prolonged activation of receptors.
Materias (normalizadas): Mitochondria
ISSN: 1673-5374
Revisión por Pares: SI
DOI: 10.4103/1673-5374.165219
Patrocinador: Ministerio de Economía y Competitividad (BFU2012- 37146)
Junta de Castilla y León (VA145U13, BIO/VA33/13, BIO103/VA45/11)
Version del Editor: http://www.nrronline.org/
Idioma: eng
URI: http://uvadoc.uva.es/handle/10324/21820
Derechos: info:eu-repo/semantics/openAccess
Aparece en las colecciones:DEP06 - Artículos de revista

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