Sfoglia per Autore "Fuente García, Miguel Ángel de la"

Items 1-20 di 69

3BP2 Deficiency impairs the response of B cells, but not T cells, to antigen receptor ligation

Fuente García, Miguel Ángel de la ; Kumar, Lalit; Lu, Bao; Geha, Raif S. (2006)
B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice

Recher, Mike; Burns, Siobhan O.; Fuente García, Miguel Ángel de la ; Volpi, Stephano; Dahlberg, Carin; Walter, Jolan E.; Moffitt, Kristin; Mathew, Divij; Honke, Nadine; Lang, Philipp A.; Patrizi, Laura; Falet, Hervé; Keszei, Marton; Mizui, Masayuki; Csizmadia, Eva; Candotti, Fabio; Nadeau, Kari; Bouma, Gerben; Delmonte, Ottavia M.; Frugoni, Francesco; Ferraz Fomini, Angela B.; Buchbinder, David; Lundequist, Emma Maria; Massaad, Michel J.; Tsokos, George C.; Hartwig, John H.; Manis, John; Terhorst, Cox; Geha, Raif S.; Snapper, Scott B.; Lang, Karl S.; Malley, Richard; Westerberg, Lisa S.; Thrasher, Adrian J.; Notarangelo, Luigi D. (2012)
Caracterización de una línea celular reportera para estudiar el mecanismo de recombinación homóloga de reparación del DNA

Peñas Pérez, Cristina (2016)
CD148 is a membrane protein tyrosine phosphatase present in all hematopoietic lineages and is involved in signal transduction on lymphocytes

Fuente García, Miguel Ángel de la ; Nicolás, Josep Maria; Freed, John H.; Palou, Eduard; Thomas, Andrew P.; Vilella, Ramón; Vives, Jordi; Gayá, Antoni (1998)
CD148, a membrane protein tyrosine phosphatase, is able to induce tyrosine phosphorylation on human lymphocytes

Palou, Eduard; Fuente García, Miguel Ángel de la ; Nicolás, Josep Maria; Vilardell, Carme; Vives, Jordi; Gayá, Antoni (1997)
CD229 (Ly9) lymphocyte cell surface receptor Interacts homophilically through Its N-Terminal domain and relocalizes to the immunological synapse

Romero, Xavier; Zapater, Nuria; Calvo, María; Kalko, Susana G.; Fuente García, Miguel Ángel de la ; Tovar, Victoria; Ockeloen, Charlotte; Pizcueta, Pilar; Engel, Pablo (2005)
CD50 (Intercellular Adhesion Molecule 3) Stimulation induces calcium mobilization and tyrmine phosphorylation through p59b and p56 in jurkat T cell line

Juan, Manel; Viñas, Odette; Pino Otín, María Rosa; Places, Lourdes; Martínez Cáceres, Eva; Barceló, Juan J.; Miralles, Agustí; Vilella, Ramón; Fuente García, Miguel Ángel de la ; Vives, Jordi; Yagüe, Jordi; Gayá, Antoni (1994)
CD84 Functions as a homophilic adhesion molecule and enhances IFN- g secretion:adhesion is mediated by Ig-like domain 1

Martín, Margarita; Romero, Xavier; Fuente García, Miguel Ángel de la ; Tovar, Victoria; Zapater, Nuria; Esplugues, Enric; Pizcueta, Pilar; Bosch, Jaime; Engel, Pablo (2001)
CD84 leukocyte antigen is a new member of the Ig superfamily

Fuente García, Miguel Ángel de la ; Pizcueta, Pilar; Nadal, Marga; Bosch, Jaime; Engel, Pablo (1997)
Cell therapy for colorectal cancer: The promise of chimeric antigen receptor (CAR)-T cells

Aparicio Fernández, Cristina ; Belver Jiménez, Marina; Enríquez, Lucía; Espeso, Francisco; Núñez Llorente, Lucía ; Sánchez García, Ana María de los Ángeles ; Fuente García, Miguel Ángel de la ; González-Vallinas Garrachón, Margarita (2021)
Characterization of endogenous Kv1.3 channel isoforms in T cells

Serna Pérez, Julia ; Peraza Pérez, Diego Alberto; Moreno Estar, Sara ; Saez, Juan J.; Gobelli, Dino Joaquin ; Simarro Grande, María ; Hivroz, Claire; López López, José Ramón ; Cidad Velasco, María Del Pilar ; Fuente García, Miguel Ángel de la ; Pérez García, María Teresa (2023)
Characterization of endogenous Kv1.3 channel isoforms in T cells.

Serna Pérez, Julia (2023)
Clinical exome analysis and targeted gene repair of the c.1354dupT variant in iPSC lines from patients with PROM1-related retinopathies exhibiting diverse phenotypes

Puertas Neyra, Kevin Louis ; Coco Martín, Rosa María ; Hernández Rodríguez, Leticia Adriána ; Gobelli, Dino Joaquin ; García Ferrer, Yenisey; Palma Vecino, Raicel; Tellería Orriols, Juan José ; Simarro Grande, María ; Fuente García, Miguel Ángel de la ; Fernández Bueno, Iván (2024)
CRISPR/CasRx proof-of-concept for RNA degradation: A future tool against RNA viruses?

Pérez San José, Diana; Fuente García, Miguel Ángel de la ; Serna Pérez, Julia ; Simarro Grande, María ; Eiros Bouza, José María ; Sanz Muñoz, Iván (2021)
Defective nuclear translocation of nuclear factor of activated T cells and extracellular signal-regulated kinase underlies deficient IL-2 gene expression in Wiskott-Aldrich syndrome

Cianferoni, Antonella; Massaad, Michel J.; Feske, Stefan; Fuente García, Miguel Ángel de la ; Gallego, María Dolores; Ramesh, Narayanaswamy; Geha, Raif S. (2005)
Desialylation of T lymphocytes overcomes the monocyte dependency of pokeweed mitogen-induced T-cell activation.

Gallart, T.; Fuente García, Miguel Ángel de la ; Barceló, Josep; Alberola-Ila, J.; Lozano, F. (1997)
Development of a novel in vitro model to study the modulatory role of the respiratory complex I in macrophage effector functions

Serrano-Lorenzo, Pablo; Gobelli, Dino; Garrido-Moraga, Rocío; Esteban Amo, María Jesús ; Orduña, Antonio; Fuente García, Miguel Ángel de la ; Martín, Miguel A.; López López, José Ramón ; Simarro Grande, María (2023)
Differential role of SLP-76 domains in T cell development and function

Kumar, Lalit; Pivniouk, Vadim; Fuente García, Miguel Ángel de la ; Laouini, Dhafer; Geha, Raif S. (2002)
Edición génica bialélica mediante inserción autocatalítica de Crispr/Cas9. deleción del canal de potasio Kv1.3 como modelo

Fernández Pelayo, Adriana Uxoa (2017)
Edición génica mediante un método CRISPR/CAS 9 autocatalítico en células humanas

Cáceres Castro, José Miguel (2018)

Sfoglia per Autore "Fuente García, Miguel Ángel de la"

3BP2 Deficiency impairs the response of B cells, but not T cells, to antigen receptor ligation ﻿

B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice ﻿

Caracterización de una línea celular reportera para estudiar el mecanismo de recombinación homóloga de reparación del DNA ﻿

CD148 is a membrane protein tyrosine phosphatase present in all hematopoietic lineages and is involved in signal transduction on lymphocytes ﻿

CD148, a membrane protein tyrosine phosphatase, is able to induce tyrosine phosphorylation on human lymphocytes ﻿

CD229 (Ly9) lymphocyte cell surface receptor Interacts homophilically through Its N-Terminal domain and relocalizes to the immunological synapse ﻿

CD50 (Intercellular Adhesion Molecule 3) Stimulation induces calcium mobilization and tyrmine phosphorylation through p59b and p56 in jurkat T cell line ﻿

CD84 Functions as a homophilic adhesion molecule and enhances IFN- g secretion:adhesion is mediated by Ig-like domain 1 ﻿

CD84 leukocyte antigen is a new member of the Ig superfamily ﻿

Cell therapy for colorectal cancer: The promise of chimeric antigen receptor (CAR)-T cells ﻿

Characterization of endogenous Kv1.3 channel isoforms in T cells ﻿

Characterization of endogenous Kv1.3 channel isoforms in T cells. ﻿

Clinical exome analysis and targeted gene repair of the c.1354dupT variant in iPSC lines from patients with PROM1-related retinopathies exhibiting diverse phenotypes ﻿

CRISPR/CasRx proof-of-concept for RNA degradation: A future tool against RNA viruses? ﻿

Defective nuclear translocation of nuclear factor of activated T cells and extracellular signal-regulated kinase underlies deficient IL-2 gene expression in Wiskott-Aldrich syndrome ﻿

Desialylation of T lymphocytes overcomes the monocyte dependency of pokeweed mitogen-induced T-cell activation. ﻿

Development of a novel in vitro model to study the modulatory role of the respiratory complex I in macrophage effector functions ﻿

Differential role of SLP-76 domains in T cell development and function ﻿

Edición génica bialélica mediante inserción autocatalítica de Crispr/Cas9. deleción del canal de potasio Kv1.3 como modelo ﻿

Edición génica mediante un método CRISPR/CAS 9 autocatalítico en células humanas ﻿

3BP2 Deficiency impairs the response of B cells, but not T cells, to antigen receptor ligation

B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice

Caracterización de una línea celular reportera para estudiar el mecanismo de recombinación homóloga de reparación del DNA

CD148 is a membrane protein tyrosine phosphatase present in all hematopoietic lineages and is involved in signal transduction on lymphocytes

CD148, a membrane protein tyrosine phosphatase, is able to induce tyrosine phosphorylation on human lymphocytes

CD229 (Ly9) lymphocyte cell surface receptor Interacts homophilically through Its N-Terminal domain and relocalizes to the immunological synapse

CD50 (Intercellular Adhesion Molecule 3) Stimulation induces calcium mobilization and tyrmine phosphorylation through p59b and p56 in jurkat T cell line

CD84 Functions as a homophilic adhesion molecule and enhances IFN- g secretion:adhesion is mediated by Ig-like domain 1

CD84 leukocyte antigen is a new member of the Ig superfamily

Cell therapy for colorectal cancer: The promise of chimeric antigen receptor (CAR)-T cells

Characterization of endogenous Kv1.3 channel isoforms in T cells

Characterization of endogenous Kv1.3 channel isoforms in T cells.

Clinical exome analysis and targeted gene repair of the c.1354dupT variant in iPSC lines from patients with PROM1-related retinopathies exhibiting diverse phenotypes

CRISPR/CasRx proof-of-concept for RNA degradation: A future tool against RNA viruses?

Defective nuclear translocation of nuclear factor of activated T cells and extracellular signal-regulated kinase underlies deficient IL-2 gene expression in Wiskott-Aldrich syndrome

Desialylation of T lymphocytes overcomes the monocyte dependency of pokeweed mitogen-induced T-cell activation.

Development of a novel in vitro model to study the modulatory role of the respiratory complex I in macrophage effector functions

Differential role of SLP-76 domains in T cell development and function

Edición génica bialélica mediante inserción autocatalítica de Crispr/Cas9. deleción del canal de potasio Kv1.3 como modelo

Edición génica mediante un método CRISPR/CAS 9 autocatalítico en células humanas